Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common
enzymatic disorder of red blood cell in human affecting more than 400 million
people worldwide. G6PD is the key regulatory enzyme in the hexose
monophosphate shunt (HMS) catalase the oxidation of glucose-6-phosphate
(G6PD) to 6-phospho gluconolacton and the production of reducing equivalents in
the form of NADPH to meet the cellular redox state and its deficiency cause
hemolytic anemia , Favism and Neonatal jaundice. In this paper we have analyzed
the G6PD gene in 73 patients with history of favism. The extracted DNA was
analyzed by polymerase
